Regulatory T cells / Ren S. Hayashi, editor.

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Bibliographic Details
Other Authors: Hayashi, Ren S.
Format: eBook
Language:English
Published: New York : Nova Science Publishers, c2011.
Series:Cell biology research progress.
Immunology and immune system disorders.
Subjects:
Online Access:Click for online access

MARC

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245 0 0 |a Regulatory T cells /  |c Ren S. Hayashi, editor. 
264 1 |a New York :  |b Nova Science Publishers,  |c c2011. 
300 |a 1 online resource. 
336 |a text  |b txt  |2 rdacontent 
337 |a computer  |b c  |2 rdamedia 
338 |a online resource  |b cr  |2 rdacarrier 
347 |a data file  |2 rda 
490 1 |a Cell biology research progress 
490 1 |a Immunology and immune system disorders 
504 |a Includes bibliographical references and index. 
505 0 |a REGULATORY T CELLS -- REGULATORY T CELLS -- Contents -- Preface -- T Helper 17 Cells Interplay with CD4+CD25highFoxp3+ Regulatory T cells in Regulating Autoimmune Inflammation -- 1. Abstract -- 2. Introduction -- 3. Comparison of the New Multi-Subset Paradigm to the old Th1-Th2 Paradigm -- 3.1. Imperfect Th1-Th2 Paradigm -- 3.2. New CD4+ Th Subsets â€? Tregs, Th17, Th9, Tfh, IL-10 IFN-Gamma-Secreting CD4+ T cells and CD4+CD28null T Cells -- 3.2.1. Tregs -- 3.2.2. Th17 -- 3.2.3. Th9 -- 3.2.4. Tfh -- 3.2.5. IL-10- IFN-gamma-secreting CD4+ T cells 
505 8 |a 3.2.6. CD4+CD28null T cells 4. Structural Characters of IL-17 Family Cytokines -- 5. Th17 Cells, a New Proinflammatory and Autoimmune CD4+ T Cell Subset -- 6. Wide Spectrum Effects of IL-17 on Various Cell Types and Tissues -- 6.1. Synovial Stromal Cells -- 6.2. Fibroblasts -- 6.3. Endothelial Cells -- 6.4. Vascular Smooth Muscle Cells (VSMCs) -- 6.5. Monocytic Cells and Macrophages -- 6.6. Structural Lung Cells -- 7. Signaling Pathways Initiated by Binding of IL-17 Cytokines and IL-17 Receptors -- 7.1. IL-17 Receptor Family 
505 8 |a 7.2. IL-17 Receptor Complex Components 7.3. IL-17 Receptor Signaling -- 8. Th17/IL-17-Mediated Inflammations -- 8.1. Experimental Autoimmune Encephalomyelitis (EAE)/Multiple Sclerosis -- 8.2. Inflammatory Skin Disease in Mice and Psoriasis in Humans -- 8.3. Inflammatory Bowel Disease (IBD) [1] -- 8.4. Experimental Arthritis/Rheumatoid Arthritis [1] -- 8.5. Atherosclerosis -- 8.6. Diabetes -- 9. Protective Roles of IL-17 in Infections -- 10. Interplay between Th17 Cells and Tregs -- 10.1. Recent Progress in Characterizing Tregs 
505 8 |a 10.2. Reciprocally Interconnected Th17 and Tregs10.3. Inhibition of Th17 by Tregs -- Conclusion -- References -- Human CD8 Tregs as Contender in Chronic Persisting Infections -- Abstract -- Introduction -- Treg Subsets, Markers and Plasticity -- CD8 Tregs in Human Disease -- Mechanism of Suppression by CD8+ Tregs -- The CCL4-CCR5 Axis as a New Player in CD8 Treg Mediated Suppression -- Evidence for Involvement of Tregs in Chronic Human Infectious Diseases -- Human CD4+ Tregs in Infectious Diseases -- Mycobacterial infections -- Parasitic infections 
505 8 |a Other bacterial infections Viral infections -- Human CD8+ Tregs in Infectious Diseases -- Mycobacterial infections -- Viral infections -- Antigen Specificity & HLA Restriction of Tregs -- Location of Tregs Suggests Significant Role in Containment of Pathogens, but Timing of Treg Activity is Crucial -- Tregs in TB Vaccination May Inhibit TB Protection: Lessons for Rational Vaccine Design -- Conclusion -- Acknowledgments -- References -- Role of Regulatory t Cells in the Treatment of Multiple Sclerosis -- Abstract -- 1. Introduction 
546 |a English. 
650 0 |a T cells. 
650 0 |a Immune response  |x Regulation. 
650 7 |a MEDICAL  |x Immunology.  |2 bisacsh 
650 7 |a Immune response  |x Regulation  |2 fast 
650 7 |a T cells  |2 fast 
700 1 |a Hayashi, Ren S. 
776 0 8 |i Print version:  |t Regulatory T cells  |d New York : Nova Science Publishers, c2011.  |z 9781617615542 (hardcover)  |w (DLC) 2010033116 
830 0 |a Cell biology research progress. 
830 0 |a Immunology and immune system disorders. 
856 4 0 |u https://ebookcentral.proquest.com/lib/holycrosscollege-ebooks/detail.action?docID=3017839  |y Click for online access 
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